Alzheimer's Disease and EMF: Learning from Henry Lai, and many others

EMF, Memory and Alzheimer's Risk>

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Alzheimer's disease is not a memory disorder; more accurately, it is a degeneration of neural and supporting tissue in the brain. It is characterized by the presence of fiber tangles within nerve cells, plaques or clusters of degenerating nerve endings, accumulation of beta-amyloid (a beta) proteins, as well as decreases in important neurotransmitters -- especially acetylcholine.

Failure of memory functions is just one effect of the process. Other cognitive and brain functions gradually deteriorate.

EMF has been studied in relationship to Alzheimer's Disease (AD) because of higher incidence of this disease in EMF-exposed workers. Several possible different, or perhaps combined mechanisms, have been proposed to explain this incidence: melatonin effects, calcium efflux and neurotransmitter alterations, breech of the blood-brain barrier, aggravation of toxic insults.

This map provides links to overviews on the topic as well as studies on various factors associated with EMF and its impact on this specific pathology.

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Occupations with EMF risk

Electric utility workers
Railway workers
Telephone workers
Machinists
Welders
Carpenters
Seamstresses
Dressmakers
Tailors

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Melatonin... helps prevent... Alzheimer’s disease... There is strong evidence from epidemiologic studies that high (≥ 10 milligauss or mG), longterm exposure to extremely low frequency (ELF, ≤ 60 Hz) magnetic fields (MF) is associated with a decrease in melatonin production.
BioInitiative Section 12

ELF-EMF levels between 0.6 and 1.2 µT have been shown to consistently block the protective effects of melatonin.
BioInitiative Section 13

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Tangles and clumps in nerves, disintegration

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Beta-amyloid plaque develops

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Shrinkage of brain tissues

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"Several mechanisms have been proposed and studied in order to explain ELF-EMF potential actions on biological systems, involving melatonin and biosynthetic enzymes in the pineal gland (melatonin hypothesis), oxidative stress, or Ca2+ efflux (release of calcium ions from a sample into a surrounding solution) in immune system cells and neurons. Other potential pathways, which may be involved in the relationship between ELF-EMF and AD include apoptosis and necrosis in brain cells, effects on biomagnetic particles reported in the human brain or differential levels of electrosensibility among the general population, but their potential nexus with AD remain unknown."
Ana Garcia, et al